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If anyone sees a case like this in his home environment, I hope to help by submitting photos of patients with a short history I hope this paper, which is part of my essay book entitled “KNOW AND OVERCOMING MEDICAL ANOMALY” SHOULD, LIKE THIS CASE DOES NOT NEED TO HAPPEN, IF PEOPLE KNOW PERKEMABNAGN DISEASE (PAOGENESIS) MEDICAL AND EFFORT THAT CAN BE EXECUTED.I hope this article can menambas insightful readers, and prevent the disorder becomes severe anomalies as reported by colleagues from the United States. June 2011 Dr Iwan suwandy, MHA Neurofibromatosis (commonly abbreviated NF; neurofibromatosis type 1 is also known as von Recklinghausen disease) is a genetically-inherited disorder in which the nerve tissue grows tumors (i.e., neurofibromas) that may be benign or may cause serious damage by compressing nerves and other tissues.Since the tumors grow where there are nerves, they can also grow in areas that are visible, causing considerable social suffering for those affected.The tumors can also grow in places that can cause other medical issues that may require them to be removed for the patient’s safety.The diagnosis of NF1 is made if any two of the following seven criteria are met: ) in chromosome 22q12.It accounts for only 10% of all cases of NF, and its frequency is lower than NF1.

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Having the initial genetic testing to determine if the mutation is recognizeable takes approximately 6 months, and then preparing the probes for the PDG testing takes approximately another 6 months.” In addition to the types of neurofibromatosis, the phakomatoses also include tuberous sclerosis, Sturge-Weber syndrome and von Hippel-Lindau disease.Therefore, if only one parent has neurofibromatosis, his or her children have a 50% chance of developing the condition as well.The severity in affected individuals can vary, this may be due to variable expressivity.It occurs following the mutation of neurofibromin on chromosome 17q11.2. Neurofibromin is a tumor suppressor gene whose function is to inhibit the p21 ras oncoprotein.In absence of this tumor suppressor’s inhibitory control on the ras oncoprotein, cellular proliferation is erratic and uncontrolled, resulting in unbalanced cellular proliferation and tumor development.

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